Specific antibody deficiency and autoinflammatory disease extend the clinical and immunological spectrum of heterozygous NFKB1 loss-of-function mutations in humans.

نویسندگان

  • Cyrill Schipp
  • Schafiq Nabhani
  • Kirsten Bienemann
  • Natalia Simanovsky
  • Shlomit Kfir-Erenfeld
  • Nathalie Assayag-Asherie
  • Prasad T Oommen
  • Shoshana Revel-Vilk
  • Andrea Hönscheid
  • Michael Gombert
  • Sebastian Ginzel
  • Daniel Schäfer
  • Hans-Jürgen Laws
  • Eitan Yefenof
  • Bernhard Fleckenstein
  • Arndt Borkhardt
  • Polina Stepensky
  • Ute Fischer
چکیده

Citation: Schipp C, Nabhani S, Bienemann K, Simanovsky N, Kfir-Erenfeld S, Assayag-Asherie N, Oommen PT, Revel-Vilk S, Hönscheid A, Gombert M, Ginzel S, Schäfer D, Laws H-J, Yefenof E, Fleckenstein B, Borkhardt A, Stepensky P, and Fischer U. Specific antibody deficiency and autoinflammatory disease extend the clinical and immunological spectrum of heterozygous NFKB1 loss-of-function mutations in humans. Haematologica. 2016; 101:xxx doi:10.3324/haematol.2016.145136

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عنوان ژورنال:
  • Haematologica

دوره 101 10  شماره 

صفحات  -

تاریخ انتشار 2016